Deletion in long arm of chromosome number 15, that leads to cause syndrome;

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#Question id: 11992

#Unit 10. Ecological Principles

Which of the following abiotic factors has the greatest influence on the metabolic rates of plants and animals? 

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#Question id: 16954

#Unit 4. Cell Communication and Cell Signaling

match the following

A. endosteal niche	1. versatile cells that both generate bone and control the differentiation of HSCs
B. osteoblasts	2. line the blood vessels and also regulate HSC differentiation
C. reticular cells	3. send processes connecting cells to bone and blood vessels, and, unexpectedly
D. sympathetic neurons	4. can control the release of hematopoietic cells from the bone marrow.

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#Question id: 14963

#Unit 2. Cellular Organization

Which antibiotics may be completely ineffective in treating a mycoplasmal infection?

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#Question id: 4686

#Unit 3. Fundamental Processes

Bacteriophage λ, has two mode of life cycle;  lytic and lysogenic.  In the lytic growth

a) a single Cro dimer is bound to OR3; this site overlaps PRM and so Cro represses that promoter

b) Repressor bound at OR1 and OR2 blocks RNA polymerase binding at PR, repressing transcription from that promoter

c) PR and PL binds RNA polymerase and directs transcription of lytic genes

d) During lytic, PRM is on while PR (and PL) are off

Find the correct combinations of lytic growth.

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#Question id: 15653

#Unit 13. Methods in Biology

You are studying a new strain of E. coli that can utilize the disaccharide melibiose very efficiently. You find that utilization depends on the enzyme melibiase, which is encoded by the gene Mel1. Mel1 is not expressed unless melibiose is present in the growth medium. You have isolated a mutation that causes constitutive melibiase activity, which you designate MelA–. P1 phage mapping experiments using a Tn5 insertion linked to Mel1 show that MelA– is not linked to Mel1. Moreover you find that when an amber suppressor is introduced into a MelA– mutant, normal melibiase regulation is restored. Classify the MelA– mutation on the basis of genetic properties,  make a proposal for the type of regulatory functions affected by the MelA– mutation?