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TLS Online TPP Program

#Question id: 7271

In drosophila, Head formation required higher Bicoid concentrations than thorax formation, because

#Unit 5. Developmental Biology
  1. The promoters of such head specific genes would have high-affinity binding sites for Bicoid protein, causing them to be activated only at extremely high concentrations of Bicoid.
  2. The promoters of such head specific genes would have low-affinity binding sites for Bicoid protein, causing them to be activated only at extremely high concentrations of Bicoid.
  3. The promoters of such head specific genes would have high- affinity binding sites for bicoid protein, causing them to be activated only at extremely low concentratrations of bicoid.
  4. The promoters of such head specific genes would have low-affinity binding sites for Bicoid protein, causing them to be activated only at extremely low concentrations of Bicoid.
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TLS Online TPP Program

#Question id: 5650

#Unit 5. Developmental Biology

Thalidomide, now banned for use as a sedative during pregnancy, was used in the early 1960s by many women in their first trimester of pregnancy. Some of these women gave birth to children with limb and organ deformities, suggesting that the drug most likely influenced ________.
TLS Online TPP Program

#Question id: 5651

#Unit 5. Developmental Biology

Growth and development of plant parts involves ________.

I) cell division to produce new cells

II) enlargement and elongation of cells

III) specialization of cells into tissues
TLS Online TPP Program

#Question id: 5652

#Unit 5. Developmental Biology

What generalization can be applied to the pole plasm of Drosophila, the P-granules of C. elegans, the yolk-free vegetal cytoplasm of Xenopus, and the localized mRNA for vasa in zebrafish?
TLS Online TPP Program

#Question id: 5653

#Unit 5. Developmental Biology

Why is meiosis required for germ cell formation, yet is never used by somatic cells?
TLS Online TPP Program

#Question id: 5654

#Unit 5. Developmental Biology

Two human disorders, Prader-Willi syndrome and Angelman syndrome, occur when a small deletion in a specific region of chromosome 15 is contributed by either the father or mother, respectively. Why does this small deletion not behave as a recessive allele for either syndrome, that is, why is its loss not made up for by the good copy of the region on chromosome 15 contributed by the other parent?
TLS Online TPP Program

#Question id: 5655

#Unit 5. Developmental Biology

What is the "acrosomal reaction"?