Following statements are regarding to the molecular basis of the coincidence model in Arabidopsis.

A. In Arabidopsis under short days, there is little overlap between CO mRNA expression and daylight.

B. CO protein does not accumulate to sufficient levels in the phloem to promote the expression of the transmissible floral stimulus and the plant remains vegetative.

C. Under long days, the peak of CO mRNA abundance (at hours 12 through 16) overlaps with daylight (sensed by phyA and cryptochrome [cry]), allowing CO protein to accumulate.

D. CO activates FLC mRNA expression in the phloem, which causes flowering when the FT protein is translocated to the apical meristem.

Which of the following is incorrect?

#Question id: 5218

#Unit 11. Evolution and Behavior

Several of the different globin genes are expressed in humans, but at different times in development. What mechanism could allow for this?

#Question id: 5219

#Unit 11. Evolution and Behavior

What is it that can be duplicated in a genome?

#Question id: 5220

#Unit 11. Evolution and Behavior

In comparing the genomes of humans and those of other higher primates, it is seen that humans have a large metacentric pair we call chromosome #2 among our 46 chromosomes, while the other primates of this group have 48 chromosomes and any pair like the human #2 pair is not present; instead the primate groups each have two pairs of midsize acrocentric chromosomes. What is the most likely explanation?

#Question id: 5221

#Unit 11. Evolution and Behavior

Unequal crossing over during Prophase I can result in one sister chromosome with a deletion and another with a duplication. A mutated form of hemoglobin, known as hemoglobin Lepore, is known in the human population. Hemoglobin Lepore has a deleted set of amino acids. If it was caused by unequal crossing over, what would be an expected consequence?

#Question id: 5222

#Unit 11. Evolution and Behavior

 When does exon shuffling occur?

#Question id: 5223

#Unit 11. Evolution and Behavior

In order to determine the probable function of a particular sequence of DNA in humans, what might be the most reasonable approach?