#Question id: 10462

During an experiments mutating the conserved cysteine in the LOV1 domain of phot1 does not affect phototropic responsiveness (the seedlings bend toward blue light) whereas the equivalent mutation in LOV2 abolishes the response. What will be the conclusion has been drawn from this experiments?

a)  LOV1 domain is secondarily responsible (primarily responsible is LOV2 domain) for kinase activation and LOV1 is not is coupled with the protein region Jα-helix that’s why it does not affect phototropic responsiveness

b) These studies have demonstrated that LOV2 domain is primarily responsible for kinase activation in response to blue light

c) This is due in part to the position of LOV2 within the phototropin molecule, where it is coupled to a protein region known as the Jα-helix

d) The function of LOV2 domain is thought to play a role in receptor dimerization by accomplishing the kinase activation in response to blue light

Which of the correct conclusion drawn from the above experiment?