TLS Online TPP Program

#Question id: 19347


Bcp1 is which type of promoter
1. Tapetum specific
2. Microspore specifc
3. Leaf specific
4. Senescence specific
Which among the following is correct combination?

#SCPH06 I Botany
  1. 1&2  
  2. 3&4  
  3. 1&4
  4. 2&3
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TLS Online TPP Program

#Question id: 419

#I Life Science/ Life Sciences Group – I-V

Biological oxidation-reduction reactions never involve:

TLS Online TPP Program

#Question id: 19691

#SCPH01 Biochemistry

The QT interval of an electrocardiogram (ECG) includes the  

TLS Online TPP Program

#Question id: 3615

#SCPH28 | Zoology

What are alleles?

TLS Online TPP Program

#Question id: 14296

#SCPH05 I Biotechnology

A two-stage chemostat system is used for production of secondary metabolite. The volume of each reactor is 0.5 m3; the flow rate of feed is 50 l h- 1. Mycelial growth occurs in the first reactor; the second reactor is used for product synthesis. The concentration of substrate in the feed is 10 gl-1. Kinetic and yield parameters for the organism are:

                                                            

Assume that product synthesis is negligible in the first reactor and growth is negligible in the second reactor. Determine cell concentrations entering the second reactor.

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TLS Online TPP Program

#Question id: 15653

#SCPH01 Biochemistry

You are studying a new strain of E. coli that can utilize the disaccharide melibiose very efficiently. You find that utilization depends on the enzyme melibiase, which is encoded by the gene Mel1. Mel1 is not expressed unless melibiose is present in the growth medium. You have isolated a mutation that causes constitutive melibiase activity, which you designate MelA–. P1 phage mapping experiments using a Tn5 insertion linked to Mel1 show that MelA– is not linked to Mel1. Moreover you find that when an amber suppressor is introduced into a MelA– mutant, normal melibiase regulation is restored. Classify the MelA– mutation on the basis of genetic properties,  make a proposal for the type of regulatory functions affected by the MelA– mutation?