#Question id: 1544
#I Life Science/ Life Sciences Group – I-V
function equivalent of MASP-2
#Question id: 14218
#SCPH05 I Biotechnology
In CSTR system at steady rate
#Question id: 7764
#SCPH01 Biochemistry
Cellular senescence
may have evolved to protect organisms against cancer. Instead of dividing out
of control, the cells die. Cellular senescence appears to be regulated by
several tumor suppressor genes, especially p53.what is the most appropriate
reason behind this,
1) Transcription
factor p53 is thought to suppress tumorigenesis by causing cell arrest and
senescence in response to short telomeres, DNA damage, and viral or external
signals to divide
2) This factor can
stop the cell cycle, cause cellular senescence in rapidly dividing cells,
instruct genes to initiate cellular apoptosis, and activate DNA repair enzymes.
3) Induction of apoptosis or cellular senescence by p53 always beneficial.
#Question id: 1547
#SCPH06 I Botany
follicular dendritic cells (FDCs) secrete CXCL13 and its receptor, CXCR5, which is expressed on B cells, function of this interaction ?
#Question id: 11959
#SCPH28 | Zoology