TLS Online TPP Program

#Question id: 1543


Effector  cytokines of TH 1 cells?

#SCPH01 Biochemistry
  1. IFN-ϒ

  2. TNF

  3. IL-21

  4. IFN-ϒ, TNF

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TLS Online TPP Program

#Question id: 2076

#SCPH05 I Biotechnology

An organism with a cell wall would most likely be unable to take in materials through:

TLS Online TPP Program

#Question id: 12683

#SCPH28 | Zoology

Which is the full complement of DNA from an Organism, carrying all the information needed to specify the structure of every protein?

TLS Online TPP Program

#Question id: 371

#SCPH05 I Biotechnology

How many milliliters of a 2.0 M solution of boric acid must be added to 600 mL of a solution of 10 mM sodium borate in order for the pH to be 9.45? (pKa of boric acid = 9.24)

TLS Online TPP Program

#Question id: 313

#SCPH28 | Zoology

What is the difference between a particle being hydrated versus being solvated?

TLS Online TPP Program

#Question id: 15152

#SCPH01 Biochemistry

In this problem we will explore some of the many ways that mutations in two different genes can interact to produce different Mendelian ratios. Consider a hypothetical insect species that has red eyes. Imagine mutations in two different unlinked genes that can, in certain combinations, block the formation of red eye pigment yielding mutants with white eyes. In principle, there are two different possible arrangements for two biochemical steps responsible for the formation of red eye pigment. The two genes might act in series such that a mutation in either gene would block the formation of red pigment. Alternatively, the two genes could act in parallel such that mutations in both genes would be required to block the formation of red pigment.
Further complexity arises from the possibility that mutations in either gene that lead to a block in enzymatic activity could be either dominant or recessive. If the crosses is made between a wild type insect with red eyes and a true breeding white eyed strain with mutations in both genes. Such considerations yield the Pathways in series with dominant mutations in both genes, determine the phenotype of the F1 progeny and the expected phenotypic ratio of red to white eyed insects in the F2.