#Question id: 7764
#SCPH01 Biochemistry
Cellular senescence
may have evolved to protect organisms against cancer. Instead of dividing out
of control, the cells die. Cellular senescence appears to be regulated by
several tumor suppressor genes, especially p53.what is the most appropriate
reason behind this,
1) Transcription
factor p53 is thought to suppress tumorigenesis by causing cell arrest and
senescence in response to short telomeres, DNA damage, and viral or external
signals to divide
2) This factor can
stop the cell cycle, cause cellular senescence in rapidly dividing cells,
instruct genes to initiate cellular apoptosis, and activate DNA repair enzymes.
3) Induction of apoptosis or cellular senescence by p53 always beneficial.
#Question id: 7764
#I Life Science/ Life Sciences Group – I-V
Cellular senescence
may have evolved to protect organisms against cancer. Instead of dividing out
of control, the cells die. Cellular senescence appears to be regulated by
several tumor suppressor genes, especially p53.what is the most appropriate
reason behind this,
1) Transcription
factor p53 is thought to suppress tumorigenesis by causing cell arrest and
senescence in response to short telomeres, DNA damage, and viral or external
signals to divide
2) This factor can
stop the cell cycle, cause cellular senescence in rapidly dividing cells,
instruct genes to initiate cellular apoptosis, and activate DNA repair enzymes.
3) Induction of apoptosis or cellular senescence by p53 always beneficial.
#Question id: 7764
#SCPH06 I Botany
Cellular senescence
may have evolved to protect organisms against cancer. Instead of dividing out
of control, the cells die. Cellular senescence appears to be regulated by
several tumor suppressor genes, especially p53.what is the most appropriate
reason behind this,
1) Transcription
factor p53 is thought to suppress tumorigenesis by causing cell arrest and
senescence in response to short telomeres, DNA damage, and viral or external
signals to divide
2) This factor can
stop the cell cycle, cause cellular senescence in rapidly dividing cells,
instruct genes to initiate cellular apoptosis, and activate DNA repair enzymes.
3) Induction of apoptosis or cellular senescence by p53 always beneficial.
#Question id: 7764
#SCPH28 | Zoology
Cellular senescence
may have evolved to protect organisms against cancer. Instead of dividing out
of control, the cells die. Cellular senescence appears to be regulated by
several tumor suppressor genes, especially p53.what is the most appropriate
reason behind this,
1) Transcription
factor p53 is thought to suppress tumorigenesis by causing cell arrest and
senescence in response to short telomeres, DNA damage, and viral or external
signals to divide
2) This factor can
stop the cell cycle, cause cellular senescence in rapidly dividing cells,
instruct genes to initiate cellular apoptosis, and activate DNA repair enzymes.
3) Induction of apoptosis or cellular senescence by p53 always beneficial.
#Question id: 7765
#SCPH01 Biochemistry
The enzyme complex or gene that maintains telomere integrity is________, which acts as an antisenescence complex. Mice and humans with its deficiencies age prematurely (Mitchell et al. 1999). Overexpressing or reactivating it in senescent cells extends longevity in mice without increasing cancer,
#Question id: 7765
#I Life Science/ Life Sciences Group – I-V
The enzyme complex or gene that maintains telomere integrity is________, which acts as an antisenescence complex. Mice and humans with its deficiencies age prematurely (Mitchell et al. 1999). Overexpressing or reactivating it in senescent cells extends longevity in mice without increasing cancer,