Given below are certain statements regarding plant-pathogen interaction;

a) P/MAMPs binds on to the pattern recognition receptor (PRR) activates the plant defence PTI

b) PTI usually stronger than ETI

c) Avr is resistance gene carry by Solanum lycopersicum (tomato) act against Avrpto effector of  pathogen Pseudomonas

d) Pathogen secreted effector molecules recognized by R-gene initiate a defence mechanism known as effector-triggered immunity

Which one of the following combination of above statement is correct?

#Question id: 1244

#Unit 4. Cell Communication and Cell Signaling

Which type of experimental evidence shows that the intrinsic GTPase activity of the Gα subunit is important for terminating effector activation?

#Question id: 1243

#Unit 4. Cell Communication and Cell Signaling

One form of receptor desensitization can be mediated by negative feedback involving phosphorylation of the receptor itself by a kinase activated downstream of a second messenger. The phosphorylated receptor likely:

#Question id: 1242

#Unit 4. Cell Communication and Cell Signaling

Rhodopsin, a light-sensitive GPCR whose role in vision is dependent on its phosphorylation status, is influenced in part by the protein arrestin.  Which of the following is TRUE with regard to rhodopsin and vision?

#Question id: 1241

#Unit 4. Cell Communication and Cell Signaling

Which of the following is a common step in the opening/closing of ion channels by acetylcholine and rhodopsin binding to their receptors?

#Question id: 1239

#Unit 4. Cell Communication and Cell Signaling

GTPases serve in many signal transduction pathways and the presence of GTP or GDP dictates whether the pathway is on or off, respectively. Which of the following statements is TRUE regarding guanine nucleotide exchange factors (GEF) and the role in these signaling pathways?

#Question id: 1238

#Unit 4. Cell Communication and Cell Signaling

Which of the following can lead to loss-of-heterozygosity in a tumor-suppressor gene?

a. deletion of the normal copy                       

b. non dysjunction during mitosis

c. somatic mutation of the normal copy

d. mitotic recombination between a wild-type and mutant allele