Practice Questions

  1. Home
  2. Free Resources
  3. Practice Questions
  4. 31858
TLS Online TPP Program

#Question id: 5031

Which era is known as the ʺage of reptilesʺ?

#11. Evolution and Behavior

  1. Cenozoic

  2. Mesozoic

  3. Paleozoic

  4. Precambrian

More Questions
TLS Online TPP Program

#Question id: 7753

#5. Developmental Biology

The best-studied type of plant autophagy, specialized organelles called autophagosomes enclose the cytoplasmic components and fuse with the vacuole, this is called
TLS Online TPP Program

#Question id: 7754

#5. Developmental Biology

In autophagy, the endoplasmic reticulum (ER) first gives rise to a cup-shaped, membranous cisterna called the
TLS Online TPP Program

#Question id: 7761

#5. Developmental Biology

Correct sets of genes are well known to be involved in aging and its prevention, and each set appears to be conserved between phyla and even kingdoms. 
a. DNA repair enzymes
b. proteins of the insulin signaling pathway
c. proteins in the mTORC1 signaling pathway
d. chromatin remodeling enzymes
TLS Online TPP Program

#Question id: 7763

#5. Developmental Biology

Two major sources of mutation are radiation and reactive oxygen species (ROS).The reactive oxygen species hypothesis for aging postulates that 
1) Senescence results from the accumulation of DNA, lipid, and protein damage inflicted by ROS of mitochondrial origin; and
2) Mitochondria of long-lived species should produce high ROS than do mitochondria of short-lived species.
3) the capacity of mitochondria to consume ROS might distinguish long-lived species from short-lived species, rather than differences in ROS generation. 
TLS Online TPP Program

#Question id: 7764

#5. Developmental Biology

Cellular senescence may have evolved to protect organisms against cancer. Instead of dividing out of control, the cells die. Cellular senescence appears to be regulated by several tumor suppressor genes, especially p53.what is the most appropriate reason behind this,

1) Transcription factor p53 is thought to suppress tumorigenesis by causing cell arrest and senescence in response to short telomeres, DNA damage, and viral or external signals to divide

2) This factor can stop the cell cycle, cause cellular senescence in rapidly dividing cells, instruct genes to initiate cellular apoptosis, and activate DNA repair enzymes.

3) Induction of apoptosis or cellular senescence by p53 always beneficial.
TLS Online TPP Program

#Question id: 7765

#5. Developmental Biology

The enzyme complex or gene that maintains telomere integrity is________, which acts as an antisenescence complex. Mice and humans with its deficiencies age prematurely (Mitchell et al. 1999). Overexpressing or reactivating it in senescent cells extends longevity in mice without increasing cancer,