TLS Online TPP Program

#Question id: 5609


Just before sperm addition, sea urchin eggs are microinjected with a calcium chelator. What event will most likely not takes place after fertilization?

#Unit 5. Developmental Biology
  1. Depolarization of the plasma membrane

  2. Elevation of PLC activity

  3. Cortical granule exocytosis

  4. An increase in intracellular pH

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TLS Online TPP Program

#Question id: 1526

#Unit 4. Cell Communication and Cell Signaling

Following statements are regarding to apoptosis.

A. In the worm apoptotic pathway, CED-3 (caspase-9 in mammals) is a protease required to destroy cell components during apoptosis.

B. CED-4 (Apaf-1) was able to accelerate the autoproteolytic cleavage and activation of purified CED-9

C. Once EGL-1 causes dissociation of the CED-4/CED-9 complex, the released CED-4 dimer joins with three other CED-4 dimers to make an octamer, which then activates CED-3

D. Many apoptotic stimuli lead to damage of the outer mitochondrial membrane, causing release into the cytosol of several proteins that stimulate apoptosis. In particular, cytochrome c released from mitochondria activates Apaf-1, which in turn activates caspase-3 and 7.

Which of the following combination is incorrect?

TLS Online TPP Program

#Question id: 3918

#Unit 3. Fundamental Processes

Which of the following is true, concerning rho?

TLS Online TPP Program

#Question id: 10921

#Unit 6. System Physiology – Plant

The mechanism of phloem translocation in angiosperms is best explained by which method?

TLS Online TPP Program

#Question id: 2199

#Unit 2. Cellular Organization

Which of the following is not an example of an ion transporter?

TLS Online TPP Program

#Question id: 7763

#Unit 5. Developmental Biology

Two major sources of mutation are radiation and reactive oxygen species (ROS).The reactive oxygen species hypothesis for aging postulates that 
1) Senescence results from the accumulation of DNA, lipid, and protein damage inflicted by ROS of mitochondrial origin; and
2) Mitochondria of long-lived species should produce high ROS than do mitochondria of short-lived species.
3) the capacity of mitochondria to consume ROS might distinguish long-lived species from short-lived species, rather than differences in ROS generation.