A hapten is best defined as being:

#Question id: 7024

#Unit 5. Developmental Biology

Which of the floral whorls is affected in apetala 3/pistillata (ap3/pi) mutants?

#Question id: 4882

#Unit 8. Inheritance Biology

In Drosophila melanogaster, cinnabar eye (cn)and vestigial wing (vg) are simple recessive traits.a A male pure line wild type eye ,vestigial wing cross with female pure line cinnabar eye, wild type wing . F1female, heterozygous for both genes, was crossed with a male with cinnabar eyes and vestigial wings. The offspring resulting from this cross are listed in below class

I Wild type

II Cinnabar eye, wild-type wing

III Wild-type eye, vestigial wing

IV Cinnabar eye, vestigial wing

Above class are recombinant progeny

#Question id: 11681

#Unit 7. System Physiology – Animal

A baby is born with a penis, a scrotum with no testes, no vagina, and XX chromosomes. This condition is referred to as hermaphroditism. Which of the following could cause this abnormality?

#Question id: 7263

#Unit 5. Developmental Biology

Following statements are regarding to the drosophila.

 A. At the completion of oogenesis, the bicoid message is anchored at the anterior end of the oocyte and the nanos message is tethered to the posterior end in translationally dormant condition.

 B. The translation inhibitors Smaug and CUP. Smaug binds to the 3’UTR of both bicoid and nanos mRNA and recruits the CUP protein that prevents the association of the message with the ribosome as well as recruiting other proteins that deadenylate the message and target it for degradation.

 C. The bicoid-Exuperantia complex is transported out of the nurse cells and into the oocyte via microtubules (Kinesin ATPase).

 D. Two other maternally provided mRNAs—caudal and hunchback are critical for patterning the anterior and posterior regions of the body plan, respectively.

 Which of the following combination is correct?

#Question id: 1234

#Unit 4. Cell Communication and Cell Signaling

Which of the following mutations would likely have a similar cancer-promoting effect as the RasD mutation?

a. mutation in Grb2 so that it cannot bind Sos

b. mutation in Sos so that it binds Ras independent of Grb2

c. mutation in GAP so that it cannot bind Ras

d. mutation in EGF receptor so that it binds GRB2 independent of EGF