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TLS Online TPP Program
#Question id: 19115
#Unit 8. Inheritance Biology
You are conducting genetic linkage studies of an autosomal recessive disease whose chromosomal location has not been firmly established. You are focused on two SSR markers that may be linked to each other and to the disease. Here are two families in which some individuals are affected:
Calculate LOD scores for linkage at θ= 0.02 between the disease and SSR43 in Family 1.
TLS Online TPP Program
#Question id: 3518
#Unit 8. Inheritance Biology
Assume that the trihybrid cross AA BB rr X aa bb RR is made in a plant species in which A and B are dominant but there is incomplete dominance between R and r. Consider the F2 progeny from this cross, and assume independent assortment. The phenotypes appear in this F2 progeny?
TLS Online TPP Program
#Question id: 21139
#Unit 12. Applied Biology
Strain improvement for the large scale antibiotics production by using methods__
I) Gene transformation
II) Mutation and selection
III) Auxotophs mutants
TLS Online TPP Program
#Question id: 1811
#Unit 4. Cell Communication and Cell Signaling
The first wave of fetal γδ T-cell receptor cells in mouse colonize the:
TLS Online TPP Program
#Question id: 15652
#Unit 13. Methods in Biology
You are studying a new strain of E. coli that can utilize the disaccharide melibiose very efficiently. You find that utilization depends on the enzyme melibiase, which is encoded by the gene Mel1. Mel1 is not expressed unless melibiose is present in the growth medium. You have isolated a mutation that causes constitutive melibiase activity, which you designate MelA–. P1 phage mapping experiments using a Tn5 insertion linked to Mel1 show that MelA– is not linked to Mel1. Moreover you find that when an amber suppressor is introduced into a MelA– mutant, normal melibiase regulation is restored. Classify the MelA– mutation in terms of its basic genetic properties,