TLS Online TPP Program
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TLS Online TPP Program
#Question id: 10910
#Unit 6. System Physiology – Plant
In early research on phloem translocation, both active and passive mechanisms were considered. All theories, both active and passive, assume an energy requirement in both sources and sinks. What will be the INCORRECT explanation of active and passive mechanism?
TLS Online TPP Program
#Question id: 1576
#Unit 4. Cell Communication and Cell Signaling
function of Properdin
TLS Online TPP Program
#Question id: 12525
#Unit 6. System Physiology – Plant
The role of ABA signaling during water stress in the guard cells in presence and absence of ABA;
a) Anion effl¬ux leads to depolarization of the plasma membrane, which drives K+ effl¬ux. The eff¬lux of ions (A– and K+) reduces the turgor pressure of guard cells, resulting in stomatal closure.
b) Both OST1 and CPKs phosphorylate and thereby activate plasma membrane anion channels, leading to ef¬flux of anions (A–). But this process can be inhibited directly by PP2Cs
c) Inhibition of PP2C activity releases the positive regulator kinase OST1 from inhibition, resulting in both phosphorylation and activation of NADPH oxidases (RBOHs)
d) RBOHs catalyze the formation of apoplastic reactive oxygen species (ROS), which triggers opening of plasma membrane Ca2+-permeable ion channels
e) The resulting elevation of cytosolic Ca2+ leads to activation of calcium-dependent protein kinases (CPKs), which can also activates the RBOH proteins further promoting Ca2+ infl¬ux into the cytosol.
Which statements belongs to the presence and absence of ABA during signalling;
I. In presence of ABA
II. In absence of ABA
Which of the following combination from the given above statements is correct?
TLS Online TPP Program
#Question id: 27674
#Unit 1. Molecules and their Interaction Relevant to Biology
Zinc Finger Motif is characterised by
TLS Online TPP Program
#Question id: 33146
#Unit 2. Cellular Organization
Which of the following protein bind to CDK4 and CDK6 monomers, blocking their interaction with cyclin D and thus rendering these G1 CDKs inactive?