The interaction between NFR and SYMRK initiates calcium spiking in the nucleus, presumably via a second messenger molecule.

Core symbiotic genes are activated.

Cytokinin signaling is initiated.

TLS Online TPP Program

#Id: 9280

#XL - Q Biochemistry

The interaction between NFR and SYMRK initiates calcium spiking in the nucleus, presumably via a second messenger molecule.

Core symbiotic genes are activated.

Cytokinin signaling is initiated.

TLS Online TPP Program

#Id: 9280

#XL - S Microbiology

The interaction between NFR and SYMRK initiates calcium spiking in the nucleus, presumably via a second messenger molecule.

Core symbiotic genes are activated.

Cytokinin signaling is initiated.

TLS Online TPP Program

#Id: 8224

#XL - Q Biochemistry

Lysosomal storage diseases:

• Hurler’s disease

• Inclusion-cell disease

TLS Online TPP Program

#Id: 8223

#XL - Q Biochemistry

Retromer

a coat protein complex specialized for endosome-to-TGN transport, recycle the M6P receptor to the TGN

TLS Online TPP Program

#Id: 8221

#XL - Q Biochemistry

The M6P receptor protein binds to M6P at pH 6.5–6.7 in the TGN lumen and releases it at pH 6, in the lumen of endosomes.

TLS Online TPP Program

#Id: 8219

#XL - Q Biochemistry

M6P residues are generated in the cis-Golgi by two Golgi-resident enzymes. 

1. N-acetylglucosamine (GlcNAc) phosphotransferase
2. Phosphodiesterase

TLS Online TPP Program

#Id: 8218

#XL - Q Biochemistry

Sorting signal that directs lysosomal hydrolases from the TGN to the late endosome is- Mannose 6-phosphate (M6P).

TLS Online TPP Program

#Id: 8217

#XL - Q Biochemistry

In selective autophagy

specific cargo is packaged into autophagosomes that tend to contain little cytosol, mediates the degradation of worn out, or unwanted, mitochondria, peroxisomes, ribosomes, and ER; used to destroy invading microbes.