Tem1-GTP activates a protein kinase cascade, which promotes the release of active Cdc14 from the nucleolus and exit from mitosis. 

TLS Online TPP Program

#Id: 5454

#Unit 7. System Physiology – Animal

The basis for the relative refractory period is the higher K+ conductance than is present at rest. 

TLS Online TPP Program

#Id: 9425

#Unit 11. Evolution and Behavior

TLS Online TPP Program

#Id: 3645

#Unit 3. Fundamental Processes

Several Class II enzymes do not interact with their bound tRNA’s anticodon.

TLS Online TPP Program

#Id: 6151

#Unit 3. Fundamental Processes

Even if cells cannot repair these lesions, there is a fail-safe mechanism that allows the replication machinery to bypass these sites of damage or tolerate the DNA damage. One mechanism of DNA damage tolerance is translesion synthesis. Although this mechanism is, as we shall see, highly error-prone and thus likely to introduce mutations, translesion synthesis spares the cell the worse fate of an incompletely replicated chromosome. A key feature of DNA damage tolerance is that the DNA lesion remains in the genome. DNA repair pathways can subsequently correct the lesion.

TLS Online TPP Program

#Id: 5790

#Unit 7. System Physiology – Animal

Thyroglobulin is a glycoprotein contains 123 tyrosine residues, but only 4–8 of these are normally incorporated into thyroid hormones.