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TLS Online TPP Program

#Question id: 2207

Why was it unexpected that transporter proteins are required for transmembrane fatty acid transport?

#Unit 2. Cellular Organization

  1. because these molecules are too small for their diffusion to be impeded by membranes

  2. because it is the cytosolic fatty acid binding-protein–fatty acid complex that is the actual transported complex

  3. because these molecules are sufficiently hydrophobic to diffuse across membranes

  4. because triglycerides carried by lipoproteins were thought to be the only source of fatty acids in the circulation

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TLS Online TPP Program

#Question id: 28849

#Unit 2. Cellular Organization

Select the correct combination of Kinesin-13?
a) It is ATPases
b) It is end-binding Proteins 
c) It is a member of the kinesin motor protein superfamily
d) It controlling the activities of proteins during the cell cycle
e) It involves to enhancing the frequency of catastrophe activity
TLS Online TPP Program

#Question id: 28850

#Unit 2. Cellular Organization

Kinesin-13 bind and curve the end of the tubulin protofilaments into which conformation?
TLS Online TPP Program

#Question id: 28851

#Unit 2. Cellular Organization

Which of the following protein greatly enhancing the frequency of catastrophes?
TLS Online TPP Program

#Question id: 28852

#Unit 2. Cellular Organization

Which of the following proteins are End-Binding Proteins?
a) XMAP215
b) Op18
c) Kinesin-13
d) stathmin
TLS Online TPP Program

#Question id: 28853

#Unit 2. Cellular Organization

Stathmin enhances that a growing microtubule will switch to the shrinking state in which condition?
a) When it increases the effective concentration of tubulin subunits that are available for polymerization 
b) When it decreases the effective concentration of tubulin subunits that are available for polymerization 
c) When stathmin is in phosphorylated condition
d) When stathmin is in dephosphorylated condition
TLS Online TPP Program

#Question id: 28854

#Unit 2. Cellular Organization

which protein promotes breakage by extracting tubulin subunits from the wall of the microtubule (also releases microtubules from MTOCs) ?