TLS Online TPP Program

#Question id: 12228


Which of the following examples best describes an ecological community?

#Unit 10. Ecological Principles
  1. The intraspecific competition of members of a brook trout population inhabiting a stream during a given year.
  2. The interactions of all the plant and animal species inhabiting a 2 hectare forest.
  3. The  material cycling  and  energy transformations between the  biotic  and  abiotic  components of  an  open meadow.
  4. The various species of barnacles competing for resources in an intertidal zone.
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TLS Online TPP Program

#Question id: 1133

#Unit 4. Cell Communication and Cell Signaling

What tertiary structure characterizes all cytokines?

TLS Online TPP Program

#Question id: 1134

#Unit 4. Cell Communication and Cell Signaling

Arrange the following proteins in the proper order in which they become activated in the MAPK signaling pathway:

a. MAP kinase                         b. MEK           c. Ras              d. Raf              e. RTK            

TLS Online TPP Program

#Question id: 1135

#Unit 4. Cell Communication and Cell Signaling

Which of the following is a direct substrate of MAP kinase?

a. TCF             b. SRF             c. SRE             d. p90RSK      e. CREB

TLS Online TPP Program

#Question id: 1136

#Unit 4. Cell Communication and Cell Signaling

Which of the following signaling pathways can be activated by activation of receptor tyrosine kinases?

a. phospholipase Cγ                b. PI-3 kinase

c. MAP kinase                         d. none of the above

TLS Online TPP Program

#Question id: 1137

#Unit 4. Cell Communication and Cell Signaling

A loss-of-function mutation in PTEN phosphatase results in

a. increased cell proliferation.            

b. decreased cell proliferation.

c. increased apoptosis.                                   

d. decreased apoptosis.

TLS Online TPP Program

#Question id: 1138

#Unit 4. Cell Communication and Cell Signaling

A loss-of-function mutation in Wnt would have a phenotype similar to a

a. gain-of-function mutation in GSK-3.                    

b. gain-of-function mutation in β-catenin.

c. loss-of-function mutation in GSK-3.                     

d. loss-of-function mutation in β-catenin.